【敬邀參加】2022.3.15 (二)12:00-13:00 美商默沙東藥廠全球臨床試驗執行處總監專題演講-臨床試驗工作剖析

【敬邀參加】美商默沙東藥廠全球臨床試驗執行處總監專題演講-臨床試驗工作剖

主題:臨床試驗工作剖析

講者:黃麗榕 執行總監

時間:2022年3月15日(二)12:00-13:00 

地點:臺北醫學大學醫學綜合大樓後棟十六樓演講廳

對象:北醫體系全體教職員生

報名連結:https://docs.google.com/forms/d/e/1FAIpQLSeWjCQyREFpifOXUIbZOlfCzokzv26TJr6Nn0n714MExk0ggQ/viewform

拇山生物醫學研究講座

主題:

Topic

Chromatin remodeling genes in gynecologic cancer progression and therapy
演講者:

Speaker

施益民 教授  Ie-Ming Shih, M.D., Ph.D.

 

Richard W. TeLinde Distinguished Professor

Director, TeLinde Gynecologic Disease Program

Co-Director, Women’s Malignancies Program
Johns Hopkins University School of Medicine

時間:

Date

111222(上午10:00~12:00

February 22, 2022, 10:00-12:00

地點:

Venue

醫學綜合大樓前棟四樓誠樸廳
4F Hall, Comprehensive Medical Building  (Front Building)
語言:

Language

中文 Chinese
主辦單位:

Organizer

醫學科學研究所

 

Abstract: 

ARID1A has emerged as a tumor suppressor gene, which is mutated in a broad spectrum of cancers, especially in those gynecologic cancers arising from ectopic or eutopic endometrium including ovarian clear cell, ovarian endometrioid and uterine endometrioid carcinomas. As a subunit of SWI/SNF chromatin remodeler, ARID1A facilitates target-specific binding of SWI/SNF complexes to chromatin, thereby altering the accessibility of chromatin to a variety of nuclear factors. In human cancer, ARID1A possesses not only features of a gatekeeper, regulating cell cycle progression, but also features of a caretaker, preventing genomic instability through DNA damage repair. Moreover, an increasing body of evidence suggests that ARID1A containing SWI/SNF complexes play a pivotal role in transcriptional regulation of genes involving in several cancer-associated  signaling pathways. In this seminar, we will discuss the mechanisms underlying how ARID1A mutations cause cancer progression and the clinical implications of synthetic lethality in developing ARID1A mutation-based therapeutics.

 

教職員請至「人才發展系統」報名:

請登入「人才發展系統」https://ehrd.tmu.edu.tw/eHRD/eHRDOrg → (左上方查詢與申請→ 課程醫科所【拇山生物醫學研究講座】 111/02/22 演講者:施益民 教授 (Johns Hopkins University School of Medicine)」(「關鍵字」可以搜尋:拇山 ) 教室確認

學生請至「學術活動網」報名:http://event.tmu.edu.tw/actnews/?Sn=4370

醫學科技學院轉譯醫學博士學位學程誠徵專任助理教授(含)以上1名(公告至111.03.16截止)

醫學科技學院轉譯醫學博士學位學程誠徵專任助理教授(含)以上1名(公告至111.03.16截止)

*學歷需求:
博士(Ph. D.)生物醫學相關領域 Biomedical Sciences or biomedicine科系畢業
*職務條件:
1.具備優良英文能力、教學及研究熱忱、具良好溝通能力
English proficiency, teaching and research enthusiasm, good communication competence
2.需有至少兩年以上博士後研究經驗或其他工作經驗
At least two years’ postdoctoral research training or related working experience
3.具有單細胞多體學、基因治療、幹細胞醫學或外泌體(exosome)研究等專長
Expertise in single cell omics、gene therapy, stem cell and exosome research
4.具參與生物製劑產業發展經驗
Experiences in biologics development and research
*需提供資料(Material needed)
除本校人力資源處網頁\聘任作業\教師聘任升等 所刊之表件外,另需提供著作錄及重要著作、可教授課程及內容大綱、過去教學之課程綱要、推薦函二封
1.個人履歷表及自傳Curriculum vitae and autobiography
2.學經歷證件影本(持外國學歷者,畢業證書需經駐外單位驗證)
Photocopy of education diploma and relative experiences (any foreign diploma should be verified in advance by the overseas office, Republic of China (Taiwan)
3.著作目錄及重要著作Publication list
4.可授之課程及內容大綱 A statement of teaching interests with course outlines
5.過去教學之課程綱要 Previous teaching portfolio/outlines and a teaching statement
6.推薦函二封Two letters of recommendation

聯絡人:熊小姐
連絡電話::(02) 2736-1661 #7602
E-MAILjuanita@tmu.edu.tw

1.申請新聘專任教師請先至系統登錄教師新聘資料本校首頁>人力資源處>教師聘任升等作業系統 (http://hr2sys.tmu.edu.tw/HRApply/default.aspx)
2.依本校人力資源處網頁>聘任作業>教師聘任升等作業規定,檢附所需表件於期限內郵寄至110臺北市信義區吳興街250號 (註明欲應徵系/所/科/學程)
For the application of our new fulltime positions, the interested applicants please register at the faculty employment and promotion operating system first (http://hr2sys.tmu.edu.tw/HRApply/default.aspx) and then make a hard copy of your curriculum vitae. Compose this hard copy, as well as the forms accompanying 
“The Procedure of Teachers ‘ Employment and Promotion ” and mail the package to the department/course you are to apply within due time.
 Please notify the unit/department, instead of any individual receiver, on the envelop for  your delivery. The address of our university.